Aluminum in vaccination-associated cognitive decline, motor neuron disease, autism

Teresa Binstock
Researcher in Developmental & Behavioral Neuroanatomy
September 28, 2009

A recent news article reported that "Cases of [Alzheimer's-like] dementia explode globally" (1). Almost simultaneously, the Journal of Inorganic Biochemistry published three articles implicating an ingredient present in many vaccines, aluminum hydroxide. First, Maryline Couette and colleagues published "Long-term persistence of vaccine-derived aluminum hydroxide is associated with chronic cognitive dysfunction" (2).

Concurrently, Shaw and Petrik reported that "Aluminum hydroxide injections lead to motor deficits and motor neuron degeneration". And despite the presumed sanctity of vaccines and vaccinations, the researchers dared conclude: "The demonstrated neurotoxicity of aluminum hydroxide and its relative ubiquity as an adjuvant suggest that greater scrutiny by the scientific community is warranted." (3) 

Aluminum hydroxide and other aluminum compounds are a component of various medications (4-5) and vaccines (eg, 6-10), wherein aluminum compounds serve as an adjuvant to hyperstimulate immunity (eg, 11).

Importantly, just as aluminum hydroxide is associated with cognitive decline and with motor neuron pathologies, and aluminum compounds are present in many vaccines, aluminum has again been found in Alzheimer's plaques in humans (12).

Dare we ask, Who among us wants to be injected with a substance associated with cognitive decline, motor neuron disease, and Alzheimer's?

We know who among us wants to inject aluminum compounds along with thimerosal (eg, 13-14), and squalene (eg, 15-18) under the guise of protecting our health. But as numbers of people with dementia, rheumatoid diseases, and autism continue to increase, perhaps time has come to desanctify vaccinationism, vaccinology, vaccinologists and their marketing representatives acting within the FDA and CDC.

Using vaccine ingredients to induce pathologies is an unwelcome burden on affected individuals and their families.


References:

1. Cases of dementia explode globally
More than 35 million live with the incurable ailment, which is expected to double every 20 years worldwide.
By Lauran Neergaard
The Associated Press, 09/21/2009
http://www.denverpost.com/ci_13383636

2. Long-term persistence of vaccine-derived aluminum hydroxide is associated with chronic cognitive dysfunction
Maryline Couette et al.
Journal of Inorganic Biochemistry (2009) in press

Macrophagic myofasciitis (MMF) is an emerging condition, characterized by specific muscle lesions assessing long-term persistence of aluminum hydroxide within macrophages at the site of previous immunization. Affected patients mainly complain of arthromyalgias, chronic fatigue, and cognitive difficulties. We designed a comprehensive battery of neuropsychological tests to prospectively delineate MMF-associated cognitive dysfunction (MACD). Compared to control patients with arthritis and chronic pain, MMF patients had pronounced and specific cognitive impairment. MACD mainly affected (i) both visual and verbal memory; (ii) executive functions, including attention, working memory, and planning; and (iii) left ear extinction at dichotic listening test. Cognitive deficits did not correlate with pain, fatigue, depression, or disease duration. Pathophysiological mechanisms underlying MACD remain to be determined. In conclusion, long-term persistence of vaccine-derived aluminum hydroxide within the body assessed by MMF is associated with cognitive dysfunction, not solely due to chronic pain, fatigue and depression.

3. Aluminum hydroxide injections lead to motor deficits and motor neuron degeneration
Christopher A. Shaw; Michael S. Petrik.
Journal of Inorganic Biochemistry (2009) in press

Gulf War Syndrome is a multi-system disorder afflicting many veterans of Western armies in the 1990–1991 Gulf War. A number of those afflicted may show neurological deficits including various cognitive dysfunctions and motor neuron disease, the latter expression virtually indistinguishable from classical amyotrophic lateral sclerosis (ALS) except for the age of onset. This ALS ‘‘cluster” represents the second such ALS cluster described in the literature to date. Possible causes of GWS include several of the adjuvants in the anthrax vaccine and others. The most likely culprit appears to be aluminum hydroxide. In an initial series of experiments, we examined the potential toxicity of aluminum hydroxide in male, outbred CD-1 mice injected subcutaneously in two equivalent-to-human doses. After sacrifice, spinal cord and motor cortex samples were examined by immunohistochemistry. Aluminum-treated mice showed significantly increased apoptosis of motor neurons and increases in reactive astrocytes and microglial proliferation within the spinal cord and cortex. Morin stain detected the presence of aluminum in the cytoplasm of motor neurons with some neurons also testing positive for the presence of hyper-phosphorylated tau protein, a pathological hallmark of various neurological diseases, including Alzheimer’s disease and frontotemporal dementia. A second series of experiments was conducted on mice injected with six doses of aluminum hydroxide. Behavioural analyses in these mice revealed significant impairments in a number of motor functions as well as diminished spatial memory capacity. The demonstrated neurotoxicity of aluminum hydroxide and its relative ubiquity as an adjuvant suggest that greater scrutiny by the scientific community is warranted.

4. Aluminum hydroxide
http://www.drugs.com/mtm/aluminum-hydroxide.html

5. Aluminum hydroxide
http://www.nlm.nih.gov/medlineplus/druginfo/meds/a699048.html

6. Vaccine Ingredients...
http://www.rense.com/general59/vvac.htm

7. Aluminum Hydroxide in Vaccines
http://www.associatedcontent.com/article/887707/aluminum_hydroxide_in_vaccines.html?cat=5

8. Aluminum-hydroxide in vaccines causes serious health problems
By Tenna Merchant
http://www.proliberty.com/observer/20071206.htm

9. Aluminum: Vaccine Education Center
http://www.chop.edu/consumer/jsp/division/generic.jsp?id=88655

10. Is Aluminum the New Thimerosal?
By Robert W. Sears
Mothering Magazine; Issue 146, January/February 2008
http://www.whale.to/vaccine/sears.html

11. The Common Vaccine Adjuvant Aluminum Hydroxide Up-Regulates Accessory Properties of Human Monocytes via an Interleukin-4-Dependent Mechanism
Marina Ulanova et al.
Infect Immun. 2001 February; 69(2): 1151–1159.
http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=97997&blobtype=pdf

12. Demonstration of aluminum in amyloid fibers in the cores of senile plaques in the brains of patients with Alzheimer’s disease
Sakae Yumoto et al.
Journal of Inorganic Biochemistry (2009) in press

Aluminum (Al) exposure has been reported to be a risk factor for Alzheimer’s disease (senile dementia of Alzheimer type), although the role of Al in the etiology of Alzheimer’s disease remains controversial. We examined the presence of Al in the Alzheimer’s brain using energy-dispersive X-ray spectroscopy combined with transmission electron microscopy (TEM-EDX). TEM-EDX analysis allows simultaneous imaging of subcellular structures with high spatial resolution and analysis of small quantities of elements contained in the same subcellular structures. We identified senile plaques by observation using TEM and detected Al in amyloid fibers in the cores of senile plaques located in the hippocampus and the temporal lobe by EDX. Phosphorus and calcium were also present in the amyloid fibers. No Al could be detected in the extracellular space in senile plaques or in the cytoplasm of nerve cells. In this study, we demonstrated colocalization of Al and beta-amyloid (Abeta) peptides in amyloid fibers in the cores of senile plaques. The results support the following possibilities in the brains of patients with Alzheimer’s disease: Al could be involved in the aggregation of Abeta peptides to form toxic fibrils; Al might induce Abeta peptides into the beta-sheet structure; and Al might facilitate iron-mediated oxidative reactions, which cause severe damage to brain tissues.

13. Hepatitis B triple series vaccine and developmental disability in US children aged 1-9 years
 Gallagher C, Goodman M. Toxicol Environ Chem 2008 90(5):997-1008.
{free online}
http://fourteenstudies.org/pdf/hep_b.pdf

"The odds of receiving EIS were approximately nine times as great for vaccinated boys... as for unvaccinated boys..., after adjustment for confounders.

14. Hepatitis B vaccination of male neonates and autism
[conference abstract as published]
CM Gallagher, MS Goodman, Graduate Program in Public
Health, Stony Brook University Medical Center, Stony Brook, NY
Annals of Epidemiology, p659
Vol. 19, No. 9 Abstracts (ACE) September 2009: 651–680
[triple the rate of autism among boys vaccinated with thimerosal versus boys not so vaccinated]

15. Vaccine A: The Covert Government Experiment That's Killing Our Soldiers--And Why GI's Are Only The First Victims
Gary Matsumoto, 2004.
http://www.amazon.com/Vaccine-Government-Experiment-Killing-Soldiers/dp/046504400X

16. Squalene & Gulf War illnesses (GWI)
http://www.rense.com/general87/mill.htm

17.  Part 1: H1N1... vaccine and human experimentation
http://tinyurl.com/m62nyd

18. Part II: Squalene laced H1N1 vaccination
http://tinyurl.com/orf6de



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